Lower Antioxidant Enzyme Contributes to Senile Cataract in Study較低的抗氧化酶，有助於老年性白內障的研究
The pathogenesis of cataract has been found to be influenced by a number of factors including oxidative stress.發病白內障已被發現受多項因素影響，包括氧化應激。 Catalase, glutathione peroxidase (GPX), and superoxide dismutase (SOD) are some of the antioxidant enzymes that protect the body from oxidative damage.過氧化氫酶，穀胱甘肽過氧化酶（ GPx ）的，和超氧化物歧化酶（ SOD ）是其中一些抗氧化酶能夠保護人體免受氧化損傷。
The present study investigates the activities of erythrocyte catalase, GPX, and SOD with respect to senile cataract (non-diabetic cataract) and osmotic cataract (diabetic cataract) in a Sri Lankan population.本研究調查活動的紅細胞過氧化氫酶， ， GPx ，超氧化物歧化酶與尊重，以老年性白內障（非糖尿病性白內障）和滲透壓白內障（糖尿病性白內障） ，在斯里蘭卡的人口。
Senile cataract (non-diabetic cataract) was associated with significantly low levels of erythrocyte catalase, GPX, and SOD when compared with osmotic cataract (diabetic cataract).老年性白內障（非糖尿病性白內障）伴有顯著水平低，紅細胞過氧化氫酶， ， GPx ，超氧化物歧化酶相比滲透壓白內障（糖尿病性白內障） 。 Positive correlations were observed between catalase and SOD (r = 0.75), catalase and GPX (r = 0.63), and SOD and GPX (r = 0.59) in subjects with senile cataracts.正相關關係，過氧化氫酶，超氧化物歧化酶（ = 0.75 ） ，過氧化氫酶和， GPx性（ r = 0.63 ） ，超氧化物歧化酶， GPx性（ r = 0.59 ） ，在學科與老年性白內障。
The results indicate that erythrocyte antioxidant enzyme levels are decreased in senile cataract as opposed to osmotic cataract.結果表明，紅細胞抗氧化酶水平下降，老年性白內障為反對滲透壓白內障。 Assays of these erythrocyte enzyme activities could provide a marker to identify individuals predisposed to senile cataract.檢測這些紅細胞酶的活動可以提供一個標記，以確定個人傾向於老年性白內障。
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